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Impact of a 2-year trial of nutritional ketosis on indices of cardiovascular disease risk in patients with type 2 diabetes.

Cardiovascular diabetology
January 1, 1970
Shaminie J Athinarayanan et al. (9 authors)
Controlled Clinical TrialJournal ArticleResearch Support, Non-U.S. Gov'tHuman StudyClinical
Study Details

Study Goal

The researchers aimed to assess the impact of a very low carbohydrate diet inducing nutritional ketosis on markers of atherosclerotic cardiovascular disease risk, particularly lipoprotein subfractions and carotid-artery intima-media thickness, in patients with type 2 diabetes.

Results Summary

The study found that the diet reduced small LDL particles (linked to CVD risk) and increased larger LDL particles, without increasing total LDL particles or ApoB, and showed no adverse progression in carotid-artery intima-media thickness. The results suggest the diet did not negatively affect CVD risk despite higher LDL-C levels.

Population

Patients with type 2 diabetes (CCI group: n=194; usual care group: n=68).

Effective Dosage

Not specified

Duration

2 years

Interactions

None mentioned

Extracted Claims (16)
InterventionDirectionEndpointPopulationDosageImpactClaim #
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
decrease
body weight
patients with type 2 diabetes (T2D)
-
resulted in improved
#1
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
improve
glycemic control
patients with type 2 diabetes (T2D)
-
resulted in improved
#2
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
decrease
multiple risk factors for cardiovascular disease (CVD)
patients with type 2 diabetes (T2D)
-
resulted in improved
#3
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
increase
low density lipoprotein cholesterol (LDL-C)
patients with type 2 diabetes (T2D)
-
resulted in
#4
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
decrease
small LDL IIIb
patients with T2D who completed 2 years of this study
23%
resulted in a 23% decrease of
#5
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
increase
large LDL I
patients with T2D who completed 2 years of this study
29%
resulted in a 29% increase of
#6
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
no change
total LDL particle concentration
patients with T2D who completed 2 years of this study
no change
resulted in
#7
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
no change
ApoB
patients with T2D who completed 2 years of this study
no change
resulted in
#8
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
decrease
small LDL subclass phenotype B
CCI participants
48.1%
was reflected by reversal of
#9
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
increase
larger cholesterol-enriched LDL particles
CCI group
-
was attributed to
#10
consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI)
no change
carotid-artery intima-media thickness (CIMT)
CCI group
no change
showed
#11
usual care (UC)
no change
carotid-artery intima-media thickness (CIMT)
UC group
no change
showed
#12
consumption of a very low carbohydrate diet with nutritional ketosis for 2 years
decrease
small LDL particles
patients with type 2 diabetes
-
lowered levels of
#13
consumption of a very low carbohydrate diet with nutritional ketosis for 2 years
increase
larger LDL particles
patients with type 2 diabetes
-
resulted in a corresponding increase in concentrations of
#14
consumption of a very low carbohydrate diet with nutritional ketosis for 2 years
increase
higher levels of plasma LDL-C
patients with type 2 diabetes
-
resulted in
#15
this dietary intervention
no change
risk of CVD
-
-
did not adversely affect
#16
Abstract

BACKGROUND: We have previously reported that in patients with type 2 diabetes (T2D) consumption of a very low carbohydrate diet capable of inducing nutritional ketosis over 2 years (continuous care intervention, CCI) resulted in improved body weight, glycemic control, and multiple risk factors for cardiovascular disease (CVD) with the exception of an increase in low density lipoprotein cholesterol (LDL-C). In the present study, we report the impact of this intervention on markers of risk for atherosclerotic cardiovascular disease (CVD), with a focus on lipoprotein subfraction particle concentrations as well as carotid-artery intima-media thickness (CIMT). METHODS: Analyses were performed in patients with T2D who completed 2 years of this study (CCI; n = 194; usual care (UC): n = 68). Lipoprotein subfraction particle concentrations were measured by ion mobility at baseline, 1, and 2 years and CIMT was measured at baseline and 2 years. Principal component analysis (PCA) was used to assess changes in independent clusters of lipoprotein particles. RESULTS: At 2 years, CCI resulted in a 23% decrease of small LDL IIIb and a 29% increase of large LDL I with no change in total LDL particle concentration or ApoB. The change in proportion of smaller and larger LDL was reflected by reversal of the small LDL subclass phenotype B in a high proportion of CCI participants (48.1%) and a shift in the principal component (PC) representing the atherogenic lipoprotein phenotype characteristic of T2D from a major to a secondary component of the total variance. The increase in LDL-C in the CCI group was mainly attributed to larger cholesterol-enriched LDL particles. CIMT showed no change in either the CCI or UC group. CONCLUSION: Consumption of a very low carbohydrate diet with nutritional ketosis for 2 years in patients with type 2 diabetes lowered levels of small LDL particles that are commonly increased in diabetic dyslipidemia and are a marker for heightened CVD risk. A corresponding increase in concentrations of larger LDL particles was responsible for higher levels of plasma LDL-C. The lack of increase in total LDL particles, ApoB, and in progression of CIMT, provide supporting evidence that this dietary intervention did not adversely affect risk of CVD.

Medical Subject Headings (MeSH)
BiomarkersCarotid Artery DiseasesCarotid Intima-Media ThicknessDiabetes Mellitus, Type 2Diet, Carbohydrate-RestrictedDyslipidemiasHeart Disease Risk FactorsHumansKetosisLipoproteins, LDLNutritional StatusNutritive ValueRisk AssessmentTime FactorsTreatment Outcome
Study Links
Quality Scores
Safety85
Efficacy90/10
Quality80/10
Citation Metrics
Total Citations40
Citations/Year8.0
Relative Citation Ratio2.69
NIH Percentile82.5%
Research Impact Scores
APT Score0.95
Weight Score1.83
Normalized Score0.86
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